A Synopsis of Ophthalmology by J. L. C. Martin-Doyle and Martin H. Kemp (Auth.)

By J. L. C. Martin-Doyle and Martin H. Kemp (Auth.)

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The point to be remembered is that no given case should be regarded as hopeless until eighteen months or two years have elapsed. In slight cases there may be no visual disability remaining. Patience is necessary, for the process of improvement may take a very long time. PATHOLOGY: The cornea in interstitial keratitis is not the real seat of the disease. The condition is really an anterior uveitis affecting the iris, ciliary body and choroid—hence posterior synechiae and ' K P ' are present, but these are not apparent clinically until the keratitis begins to subside, for the corneal infiltration prevents a good view of the inner structures of the eye.

Glandular enlargement, especially in the posterior triangles of the neck. e. The limbs should be examined for periosteal nodules and synovitis of joints. The Wassermann reaction will settle the diagnosis. Note: It must be remembered that very rarely a tuberculous iritis may be associated with a keratitis closely resembling interstitial keratitis. TREATMENT: a. Antisyphilitic treatment is most disappointing in interstitial keratitis and does not influence the course of the disease owing to the non-vascularity of the cornea.

Blood Supply: The blood supply of the posterior part of the uveal tract is through the short posterior ciliary arteries. The anterior part of the tract derives its supply from the anterior ciliary arteries and the two long posterior ciliary arteries. The ciliary body is supplied by the long posterior arteries which form a circle at the root of the iris, called the greater arterial circle of the iris. The anterior ciliary arteries reach the globe by the recti muscles passing through the sclera posterior to the corneosclerotic junction.

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